Reflect on the Adenylyl Cyclase signaling cascade. Consider this cascade and the intracellular impacts that it has on a liver hepatacyte when [glucagon] is high. Every statement below accurately describes events that would occur except for one. Please select the FALSE statement. The Gs alpha will activate the adenylyl cyclase effector and the second messenger will be formed. Reflection on the Adenylyl Cyclase Signaling Cascade in Liver Hepatacytes When Glucagon is Hig

Reflection on Liver Hepatacytes’ Adenylyl Cyclase Signing Cascade when Glucagon Is High
Adenylylcyclase is an important component of intracellular reactions in a liver-hepatacyte that occurs when glucagon levels are high. This cascade begins with glucagon. This is the first messenger. This activation activates a G-protein signal transducer which then activates Gs alpha subunit and finally the adenylylcyclase efector. The second messenger is activated by the effector, which produces cAMP. The PKA activation leads to the phosphorylation and activation of the enzymes fructose-1,6-bisphosphatase (FBPase) and phosphofructokinase-2 (PFK-2), resulting in the dephosphorylation of PFK-2 and elevated levels of fructose-2,6-bisophosphate (F2,6BP) (Komarova, 2020). Concerning the Adenylyl Cyclase Signaling Cascade in liver Hepatacytes, when glucagon levels are high, FALSE statements state that the signaling sequence will result in the phosphorylation or inactivation pyruvate Kinase. PKA activation results in the phosphorylation enzymes such as FBPase or PFK-2. However, it doesn’t lead to the phosphorylation or inactivation pyruvate kinase. (Jia, 2019,). Cont…
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